1. AQP4‑NMOSD optic neuritis (most likely)
2. MOGAD optic neuritis (usually more anterior, optic sheath oedema)
3. Multiple sclerosis optic neuritis (short segment, unilateral)
4. Granulomatous/infective optic neuritis (sarcoid, TB, syphilis)
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Explanation:
Lost appendicoliths (as well as gall stones) can cause delayed abscesses years after the surgical procedure. Due to a tilted position of the patient during surgery, the lost stones may “travel” towards the upper abdomen and can cause abscesses in the Morison pouch.
Therapy consists of surgical or interventional removal of the stone and drainage of the abscess.
References:
– Singh AK, Hahn PF, Gervais D, Vijayraghavan G, Mueller PR. Dropped appendicolith: CT findings and implications for management. AJR Am J Roentgenol. 2008 Mar;190(3):707-11. doi: 10.2214/AJR.07.2917. PMID: 18287442.
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Explanation:
There are areas of bone marrow oedema in the left SI joint, with a small amount of joint space fluid in the upper segments, along with increased subchondral sclerosis and erosions. No features of capsulitis, no evident backfill or ankylosis visible. Joint space enhancement cannot be confirmed—this was a non-contrast study.
Explanation:
To be eligible for the ASAS criteria, patients must have chronic back pain (lasting more than three months) with onset before age 45.
They can then be classified into one of two groups: the “imaging arm” if they have evidence of sacroiliitis (on X-ray or MRI) and at least one other feature of spondyloarthritis, or the “clinical arm” if they are HLA-B27 positive and have two or more other features of spondyloarthritis (e.g. uveitis, enthesitis, dactylitis, positive family history for axSPA, psoriasis, increased C-reactive protein level, etc).
Dumbbell-shaped lesion in third web space near the metatarsophalangeal joint with isointensity on T1 and hypo to isointensity and T2 sequences and inhomogeneous contrast enhancement
Morton’s neuroma in third web space
Adhesive capsulitis
There is a thickening and edema of the inferior joint capsule in the axillary recess which is characteristic of adhesive capsulitis.
Additionally, there is an interstitial tear of supraspinatus tendon.
Adhesive capsulitis, also known as frozen shoulder, is an inflammatory condition characterized by shoulder stiffness, pain, and significant loss of passive range of motion.
The joint capsule exhibits inflammation, thickening and fibrosis, leading to pain and loss of ROM.
In most cases, adhesive capsulitis is a self-limited disease with high spontaneous recovery rates within 18 to 30 months.
Diarrhea, postprandial dizziness and weakness
Weight loss of 20 kgs in the last 2 years
CT imaging was acquired
Mesenteric mass with calcifications surrounded by tethered small bowel loops
Retroperitoneal lymphadenopathy
Hypodense hepatic lesion, most likely metastasis
Mesenteric, retroperitoneal lymph node and hepatic metastasis of small bowel neuroendocrine tumor (NET)
Most commonly, this tumor originates in the terminal ileum, but in many cases, the submucosal primary lesion can’t be identified on CT
The mesenteric mass shows both calcifications and tethering of the surrounding bowels due to the characteristic desmoplastic reaction of the mesentery
For further work-up, the radiotracer Ga-68 DOTATATE is shown to be the most accurate at identifying NET
40-year-old female:
with chronic medial side knee pain.
Pellegrini-Stieda lesion
There is an ossified lesion adjacent to medial femoral condyle, at the insertion of medial collateral ligament (MCL).
MCL fibers are intact.
No signs of osteoarthritis.
Slight degenerative changes within the body of medial meniscus.
Pellegrini-Stieda lesions are defined as post-traumatic ossifications of the medial collateral ligament (MCL) at or near its proximal insertion on the medial femoral condyle.
Pellegrini-Stieda disease (or syndrome) is defined as the combination of the radiographic findings and concomitant medial knee joint pain or restricted range of motion.
